PRION2013 CONGRESSIONAL ABSTRACTS CWD


Sunday, August 25, 2013

Prion2013 Chronic Wasting Disease CWD risk factors, humans, domestic cats, blood, and mother to offspring transmission

http://chronic-wasting-disease.blogspot.com/2013/08/prion2013-chronic-wasting-disease-cwd.html


Friday, August 09, 2013

CWD TSE prion, plants, vegetables, and the potential for environmental contamination

http://chronic-wasting-disease.blogspot.com/2013/08/cwd-tse-prion-plants-vegetables-and.html


Thursday, August 08, 2013

Characterization of the first case of naturally occurring chronic wasting disease in a captive red deer (Cervus elaphus) in North America

http://chronic-wasting-disease.blogspot.com/2013/08/characterization-of-first-case-of.html


Sunday, September 01, 2013

hunting over gut piles and CWD TSE prion disease

http://chronic-wasting-disease.blogspot.com/2013/09/hunting-over-gut-piles-and-cwd-tse.html


Wednesday, September 04, 2013

cwd - cervid captive livestock escapes, loose and on the run in the wild...

http://chronic-wasting-disease.blogspot.com/2013/09/cwd-cervid-captive-livestock-escapes.html


Tuesday, September 10, 2013

Review and Updates of the USDA-APHIS Veterinary Services (VS) National Chronice Wasting Disease (CWD) Program 2012-2013

http://chronic-wasting-disease.blogspot.com/2013/09/review-and-updates-of-usda-aphis.html



Sunday, August 11, 2013

Creutzfeldt-Jakob Disease CJD cases rising North America updated report August 2013

Creutzfeldt-Jakob Disease CJD cases rising North America with Canada seeing an extreme increase of 48% between 2008 and 2010

http://creutzfeldt-jakob-disease.blogspot.com/2013/08/creutzfeldt-jakob-disease-cjd-cases.html



Friday, August 16, 2013

*** Creutzfeldt-Jakob disease (CJD) biannual update August 2013 U.K. and Contaminated blood products induce a highly atypical prion disease devoid of PrPres in primates

http://creutzfeldt-jakob-disease.blogspot.com/2013/08/creutzfeldt-jakob-disease-cjd-biannual.html



Sunday, September 08, 2013

Iatrogenic Creutzfeldt-Jakob disease via surgical instruments and decontamination possibilities for the TSE prion

http://creutzfeldt-jakob-disease.blogspot.com/2013/09/iatrogenic-creutzfeldt-jakob-disease.html



*** The potential impact of prion diseases on human health was greatly magnified by the recognition that interspecies transfer of BSE to humans by beef ingestion resulted in vCJD. While changes in animal feed constituents and slaughter practices appear to have curtailed vCJD, there is concern that CWD of free-ranging deer and elk in the U.S. might also cross the species barrier. Thus, consuming venison could be a source of human prion disease. Whether BSE and CWD represent interspecies scrapie transfer or are newly arisen prion diseases is unknown. Therefore, the possibility of transmission of prion disease through other food animals cannot be ruled out. There is evidence that vCJD can be transmitted through blood transfusion. There is likely a pool of unknown size of asymptomatic individuals infected with vCJD, and there may be asymptomatic individuals infected with the CWD equivalent. These circumstances represent a potential threat to blood, blood products, and plasma supplies.


http://cdmrp.army.mil/prevfunded/nprp/NPRP_Summit_Final_Report.pdf



kind regards, terry

Had a grandpaw in law die of CJD 2 years ago. They have no idea how he got it and really didn't know exactly what he had until he passed away. Doctors told us 1 in 1,000,000 get what he had. Went from healthy to dead in 2 months.

As far as I know it is perfectly legal to hunt in northern states that have had CWD proven infestation and to bring back to Alabama (or any other southern state) the head for mounting by a taxidermist. The taxidermist skins the head and then cuts the antlers from the skull, using a saw that would expose the prions to the taxidermist and any clients that might come into the shop. A butcher shop processing the animal would have the same exposure. Hundreds of deer each year are mounted by taxidermist and the meat processed by a butcher. The prions can then be carried afield and could/would potentially contaminate all the hunting areas the hunters go to. Heat nor cold temperatures kill the prions. Yet there hasn't been a single (as far as I know) reported CWD case because of this type action. Either CWD isn't as easily transferred and exposed or something else is up with the CWD. Nobody I have ever talked with has an answer to my concerns/questions.

I spend a lot of time studying CWD and have also not heard of a single transmission of CWD by bring an infected animal head/carcass into another state.

Just something to think about--back when England had that BSE (mad cow) prion outbreak in the cows, only a few hundred people got it. When you look at how many cows had it, and estimate how many were slaughtered and put into the food supply before they knew what was happening, in all probability all the beef-eaters in England got some of that meat. Most of them never showed any symptoms. In all liklihood, a few of us will eventually die from eating CWD carrying deer, but most of us are probably immune.

BSK, any opinion you have on my question/statement above? Or any other biologist?

Best estimate were 35 million people ate MCD infected meat. Only 218 people got the disease. That's an exceptionally low transmission rate.

Considering Europe's experience showed that it was possible to contract CJDv from eating infected beef, it has to be a concern that people might contract CJD from eating infected deer. However, considering it was possible to identify the variant of CJD for those who contracted the disease from eating infected beef, most likely the same would be true for people infected by eating deer, and no one who has CJD has displayed a variant variety of the prion to date.

One of the problems with deer and CJD (mad cow) human transmission is that mad-cow was caused by cows being fed cows (cannibal cows). The bad prion developed in cows from the cannibalism just like "Kuru" developed in people in New Guinea. We ate cannibal cow and got mad-cow. In New Guinea they ate other cannibals and got "kuru".
The symptoms of CJDv may be the same as mad-cow but the prion disease is likely caused by something we have yet to discover. Unless there are cannibal deer running around and we dont know it.

Not exactly. You bring a head, spine, bone back from through KY or TN from a CWD state and they'll fine the heck out of you and take your deer. Arkansas and MO are the same.

Heads must be completely caped, antlers removed. Meat de-boned.

I think a comparison of CWD to scrapie in sheep and goats would be a better comparison then comparing CWD to BSE based on what is known so far.

yall look at the post flounder has made . lol


he don't want us eating deer i think . lol

Swamper I know a few taxidermist and all of them mount out of state deer, lots of them from northern states. The taxidermist have been doing it for years. If the prions are the carriers of CWD disease and they are immune from cold or heat then that stuff would have infected the southern states a long time ago, quicker than Sherman went thru Georgia.

My dad has a client that this personally happened to. I'm not positive on the state they had hunted but I believe it was Colorado. On their way home, they were stopped in KY with two caped heads. I can't remember exact dollar amounts, but the heads were confiscated and they were fined out the a$$! I'll talk to him and get the whole story but, if I'm not mistaken, it was around 5k.

CWD came from scrapie infected sheep at the Foothills Wildlife Research Station in Colorado. It IS scrapie, only in deer.

NOTHING is know of the period of time for CWD to show up in infected humans. Kuru has an incubation period of something like 20-30 years if I remember correctly(?)....

I think that is the point of no brain, spine, ect. Apparently, and BSK or Fred might can clear it up, the disease is infected in those areas of the deer. I just know the laws and there has to be a reason you better not get caught with skull, spine and some states any bone except a cleaned skull plate.

That is one theory but so far I haven't seen where it has satisfied Koch's postulates.



Where and How Did CWD Originate?

The origin of CWD is unknown, and it may never be possible to definitively determine how or when CWD arose. It was first recognized as a syndrome in captive mule deer held in wildlife research facilities in Colorado in the late 1960s, but it was not identified as a TSE until the 1970s. Computer modeling suggests the disease may have been present in free-ranging populations of mule deer for more than 40 years.

Scrapie, a TSE of domestic sheep, has been recognized in the United States since 1947, and it is possible that CWD was derived from scrapie. It is possible, though never proven, that deer came into contact with scrapie-infected sheep either on shared pastures or in captivity somewhere along the front range of the Rocky Mountains, where high levels of sheep grazing occurred in the early 1900s.

It may be possible that CWD is a spontaneous TSE that arose in deer in the wild or in captivity and has biological features promoting transmission to other deer and elk.

© Chronic Wasting Disease Alliance

horseshit ...

CWD originated in the research pens at Foothills Wildlife Research Station at Ft Collins in the early 60's . Originally wild mule deer does were captured and placed in pens that had held scrappie infected sheep(this is a fact which early researchers state but has been rewritten by later folks to take the blame off the research station. After birthing some excess does were released into the wild near Ft Collins, which became the epicenter of the disease. Wonder why???? The fawns/does were kept for research. Excess deer were sent/traded whatever to several zoos, other research stations, private enclosures, etc. Early on one could trace every known instance of CWD straight back to Ft Collins and nowhere else.

originated in the wild...horseshit.

Bad science.

First of all...I have never seen anywhere that anyone denies that CWD may have come from the pens at Fort Collins. If someone did prove that it came from the pens at Fort Collins that would be a great career boost for them so why would they not publish it? Your whole conspiracy theory about a cover up is idiotic. Why would anyone want to cover that up...nothing was done wrong.

Secondly, the deer in the pens were captured in many cases from the population outside the pen. So perhaps the disease originated in the pens but if it originated outside the pens then it would not be surprising that it was brought in. Some of the epidemiology suggests it had been in the area for some time before being detected. Even then the disease could be related to scrapie because the sheep have been grazed in that area for many years.

As far as shipping the disease to zoos, etc....of course they all are traced back to Fort Collins....they weren't shipping wild deer out but the deer they had captive. That proves nothing but that the disease originated in the region which everyone agrees with.

Reaching conclusions that can't be substantiated is the basis of bad science.

I don't care what you eat. I am a meat eater and firm believer in our 2nd and 1st amendment rights. ...


kind regards,
terry